Preliminary studies show conolidine may well inhibit certain ion channels, minimizing neuronal excitability and restricting ache alerts. This system is particularly related in neuropathic suffering, wherever abnormal signaling brings about persistent discomfort. Additionally, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to agony perception. Computatio... https://johna703dbv2.wikifrontier.com/user